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Hemolytic anemia: why red blood cells are destroyed and how to recognize it

Other names: Гемолитическая анемия, гемолиз, разрушение эритроцитов, аутоиммунная гемолитическая анемия, желтуха при анемии, повышенный билирубин и низкий гемоглобин

Hemolytic anemia is a group of conditions in which red blood cells are destroyed prematurely. Normally, a red cell lives about one hundred and twenty days; with hemolysis, it lasts a matter of weeks or even days. The bone marrow tries to compensate for the loss and releases young forms into the blood, so reticulocytes are increased. Hemoglobin is released from the disintegrating red blood cells, it turns into bilirubin, and the skin and whites of the eyes acquire a yellowish tint, and the urine may darken. The causes are congenital, when the defect lies in the cell itself, and acquired, when red blood cells are destroyed by the immune system, infection, medication or mechanical obstacle.

🧾 МКБ-10: D59 🏥 Where it is treated: 3 Red blood cells live shorterYellowish and dark urineReticulocytes are elevated
👨‍⚕️ Which doctor
Hematologist, therapist, pediatrician
🔬 Diagnostics
Complete blood count, reticulocytes, bilirubin, LDH, haptoglobin, Coombs test, ultrasound of the spleen
💊 Treatment
Depends on the cause: glucocorticoids and immunosuppression in the autoimmune form, drug withdrawal, folic acid, in some cases, removal of the spleen
📈 Prognosis
Various: from complete recovery to lifelong observation
⚠️ At risk
Heredity, autoimmune diseases, infections, certain medications, valve prostheses
⏱ When to see a doctor
Urgent for rapidly increasing pallor, jaundice and dark urine

🚨 See a doctor urgently

With these signs do not wait for a scheduled appointment — the condition requires emergency care.

  • Быстро нарастающая бледность и слабость за один-два дня
  • Тёмная, почти коричневая моча
  • Выраженная желтизна кожи и белков глаз
  • Одышка и сердцебиение в покое, обморок
  • Боль в пояснице или животе на фоне желтухи
  • Желтуха у новорождённого в первые сутки жизни

Types of hemolysis

Hemolysis is divided by location and cause. Intracellular occurs in the spleen, where macrophages capture altered red blood cells; this form is accompanied by an enlarged spleen and moderate jaundice. Intravascular hemolysis means the destruction of cells directly in the bloodstream: free hemoglobin enters the plasma, binds to haptoglobin, and when it is depleted, it is excreted by the kidneys, turning the urine dark. For this reason, congenital forms with a defect in the membrane, enzyme or hemoglobin itself are distinguished and acquired forms, among which autoimmune ones are most common.

  • Intracellular hemolysis in the spleen
  • Intravascular hemolysis with hemoglobin in plasma and urine
  • Hereditary spherocytosis and membrane defects
  • Red blood cell enzyme deficiency
  • Thalassemias and sickle cell disease
  • Autoimmune forms with warm or cold antibodies

Reasons and provocateurs

Autoimmune hemolytic anemia can be independent or accompany systemic diseases, lymphoproliferative diseases and infections. Some drugs can trigger immune destruction of red blood cells, so the doctor always checks the list of medications taken. With a hereditary deficiency of certain enzymes, an attack is provoked by an infection, certain medications and certain foods, such as faba beans. Mechanical failure occurs in artificial heart valves and in some small vessel conditions. In newborns, the cause is incompatibility between the blood of mother and child.

  • Autoimmune diseases and lymphomas
  • Viral and bacterial infections
  • Selected medications
  • Hereditary membrane and enzyme defects
  • Mechanical damage to the prosthetic valve
  • Incompatibility of maternal and fetal blood
  • Transfusion of incompatible blood

Symptoms

The picture consists of manifestations of anemia and signs of hemolysis itself. Common complaints are weakness, dizziness, shortness of breath, palpitations, decreased exercise tolerance. The specific part is yellowness of the skin and sclera without itching, dark urine, enlarged spleen and a feeling of heaviness in the left hypochondrium. In chronic cases, gallstones often form in the gallbladder due to excess bilirubin. With acute massive hemolysis, the condition worsens quickly: chills, fever, lower back pain, severe weakness appear, and this situation requires emergency help.

  • Weakness, dizziness, shortness of breath
  • Yellowness of the skin and whites of the eyes
  • Darkening of urine
  • Enlarged spleen
  • Gallbladder stones with a long course
  • Chills, fever and lower back pain in acute crisis

Diagnostics

First, the fact of hemolysis is confirmed: in a general blood test, hemoglobin is reduced, reticulocytes are increased, in biochemistry indirect bilirubin and LDH increase, and haptoglobin, on the contrary, decreases. Then they look for the reason. The direct Coombs test determines whether there are antibodies on red blood cells and separates autoimmune forms from others. A blood smear shows characteristic changes in cell shape. According to indications, hemoglobin electrophoresis is performed if thalassemia is suspected, enzyme and osmotic resistance studies, ultrasound of the spleen and gallbladder.

  • General blood test and smear
  • Reticulocytes
  • Indirect bilirubin, LDH, haptoglobin
  • Direct Coombs test
  • Hemoglobin electrophoresis according to indications
  • Ultrasound of the spleen and gallbladder
  • Screening for infections and autoimmune diseases

Treatment and observation

Treatment depends entirely on the cause, and there is no universal treatment. In the autoimmune form, glucocorticoids serve as the basis; if the effect is insufficient, immunosuppressive or anti-B-cell drugs are added. If hemolysis is caused by the drug, it is discontinued. In hereditary forms, it is important to prevent provoking factors and take folic acid, and in severe cases, removal of the spleen is discussed with mandatory preliminary vaccination. Red blood cell transfusion is carried out according to strict indications. Iron supplements without a confirmed deficiency are not prescribed for hemolysis.

  • Glucocorticoids for autoimmune form
  • Immunosuppressive and anti-B cell drugs
  • Discontinuation of the culprit drug
  • Folic acid for chronic hemolysis
  • Splenectomy according to indications after vaccination
  • Red blood cell transfusion for severe anemia
  • Do not take iron without a proven deficiency

Services and prices for this diagnosis

Based on official price lists of Tashkent clinics. The exact cost is determined after examination.

Frequently asked questions: Hemolytic anemia

How to distinguish hemolytic anemia from iron deficiency?+
According to analyses. With iron deficiency, reticulocytes are low, ferritin is reduced, and bilirubin is normal. With hemolysis, reticulocytes are elevated, indirect bilirubin and LDH increase, haptoglobin decreases, and iron reserves are usually not reduced. Therefore, it is impossible to prescribe iron without examination.
Why does the skin turn yellow with this anemia?+
When red blood cells are destroyed, hemoglobin is released, which is converted into indirect bilirubin. The liver does not have time to process it, bilirubin accumulates in the blood and stains the skin and whites of the eyes. Unlike liver diseases, there is usually no itching of the skin.
Is it necessary to remove the spleen?+
No. Surgery is considered only for certain forms and insufficient effect of drug treatment. Before it, it is necessary to vaccinate against pneumococcus, meningococcus and Haemophilus influenzae, since after removal of the spleen the risk of severe infections increases.
Is it possible to plan a pregnancy?+
Yes, but always together with a hematologist and obstetrician-gynecologist. Pregnancy can provoke an exacerbation of hemolysis, so planning in a stable period, regular monitoring of tests and a pre-agreed list of safe drugs are needed.
Does hemolysis always mean lifelong treatment?+
No. If hemolysis is caused by an infection or a drug, once the cause is eliminated, the blood is completely restored. Hereditary and chronic autoimmune forms require long-term observation, but they are also well controlled with selected therapy.

The information on this page is for reference only and does not replace a doctor consultation. Only a qualified specialist can make a diagnosis and prescribe treatment after an in-person examination.

Where it is treated hemolytic anemia в Ташкенте

Разобраться, почему разрушаются эритроциты, можно только по набору анализов, и делать это должен гематолог вместе с терапевтом. Clinics Ташкента, где доступны нужные исследования:

Tashkent, Chilanzar district, st. U. Nasyra, 138d
M Olmazor 🚶 2.6 km
M Chilonzor 🚶 2.7 km
M O'zgarish 🚶 2.8 km
🚌 Nearest bus stop 🚶 80 m · buses: 38, 40, 57, 58
Mon–Fri:09:00–17:00
Closed now
Tashkent, Chilanzar district, st. Katartal, 42d
M Chilonzor 🚶 550 m
M Mirzo Ulug'bek 🚶 800 m
M Olmazor 🚶 1.5 km
🚌 Nearest bus stop 🚶 60 m · buses: 34, 56
Mon–Fri:09:00–17:00
Closed now
Tashkent city, Shaykhantokhur district, st. Kukcha Darvoza-42
M Chorsu 🚶 1.5 km
M Tinchlik 🚶 1.9 km
M Milliy bog' 🚶 2.1 km
🚌 Nearest bus stop 🚶 190 m · buses: 20, 27, 35, 46, 53
Пн–Sun:00:00–24:00
Open now

ICD-10 code

Official international classification codes — these are used in medical records and statistics.

Other diseases: Hematology

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