dr hasan
🏥kliniki*

Kidney amyloidosis: swelling, protein in urine and how to diagnose

Other names: Амилоидоз почек, амилоидная нефропатия, AA-амилоидоз, AL-амилоидоз почек, отложение амилоида в почках, вторичный амилоидоз

Kidney amyloidosis is a disease in which amyloid is deposited in the kidney tissue: an abnormal protein with a special structure that the body cannot break down. The deposits gradually replace the normal structures of the glomeruli, and the kidney begins to lose protein in the urine. The typical picture is increasing edema, massive proteinuria and a decrease in protein levels in the blood. Amyloidosis is rarely isolated: more often it is part of a systemic process associated with chronic inflammation, blood diseases or hereditary syndromes. The diagnosis is confirmed only by a biopsy with a special stain, so if there is persistent protein in the urine, the examination cannot be postponed.

🧾 МКБ-10: E85.4 🏥 Where it is treated: 6 Massive protein in urineDiagnosis - by biopsyTreat the cause and support the kidneys
👨‍⚕️ Which doctor
Nephrologist, rheumatologist, hematologist
🔬 Diagnostics
Protein in urine and daily proteinuria, kidney biopsy, creatinine, blood protein fractions
💊 Treatment
Treatment of the underlying disease, nephroprotection, with the AL form - hematological therapy
📈 Prognosis
Serious, but depends on the form and timing of treatment
⚠️ At risk
Chronic inflammatory and rheumatic diseases, tuberculosis, bronchiectasis, myeloma, periodic illness
⏱ When to see a doctor
Scheduled, examination in the coming weeks

🚨 See a doctor urgently

With these signs do not wait for a scheduled appointment — the condition requires emergency care.

  • Быстро нарастающие отёки ног, живота и лица
  • Одышка в покое или в положении лёжа
  • Резкое уменьшение количества мочи
  • Пенистая моча, сохраняющаяся неделями
  • Обмороки, головокружение при вставании, очень низкое давление
  • Необъяснимая потеря веса и увеличение языка

Forms of amyloidosis and where amyloid comes from

Amyloid is not one protein, but a whole family: depending on the precursor protein, different types of the disease are distinguished. AA amyloidosis develops against the background of long-term inflammation, when the liver produces acute phase protein for years. AL amyloidosis is associated with an abnormal clone of plasma cells that produce immunoglobulin light chains and is a blood disease. There are also hereditary forms, including periodic disease, common among the peoples of the Mediterranean and the Middle East. The type of amyloid determines treatment, so efforts are made to determine it accurately.

  • AA amyloidosis is the outcome of chronic inflammation
  • AL amyloidosis - associated with plasma cell disease
  • Hereditary forms, including periodic illness
  • Dialysis amyloidosis with long-term hemodialysis
  • Senile systemic amyloidosis

Causes and risk factors

The AA form is provoked by diseases in which inflammation lasts for years: rheumatoid arthritis, ankylosing spondylitis, inflammatory bowel diseases, chronic suppurative processes, tuberculosis, bronchiectasis, osteomyelitis. Periodic illness without treatment is also often complicated by renal amyloidosis. The AL form is associated with monoclonal gammopathy and myeloma. The longer and more active the underlying disease is, the higher the risk.

  • Rheumatoid arthritis and other rheumatic diseases
  • Periodic disease (familial Mediterranean fever)
  • Chronic purulent processes, osteomyelitis, bronchiectasis
  • Tuberculosis
  • Inflammatory bowel diseases
  • Myeloma and monoclonal gammopathy

Symptoms

The first and for a long time the only sign is protein in the urine, discovered by chance. Then nephrotic syndrome develops: severe edema, foamy urine, decreased blood protein and increased cholesterol. Since amyloid is deposited in other organs, signs of damage to the heart, intestines and nerves may occur. Especially characteristic of the AL form are enlarged tongue, bruises around the eyes and severe weakness.

  • Swelling of the legs, lower back, face, fluid accumulation in the abdomen
  • Foamy urine and massive proteinuria
  • Weakness, decreased blood pressure, dizziness when standing up
  • Shortness of breath due to heart damage
  • Persistent diarrhea or constipation
  • Numbness and burning in the feet and hands

Diagnostics

The examination begins with a urine test and assessment of daily protein loss, then creatinine, total protein and blood albumin, and lipids are determined. The next task is to prove the presence of amyloid and establish its type. To do this, a biopsy of the kidney or other available tissue is performed with Congo red staining. Additionally, they look for the cause: they examine the joints, lungs, intestines, and conduct tests for monoclonal protein. All this is done in stages, the nephrologist selects the plan.

  • General urine analysis and daily proteinuria
  • Creatinine, urea, GFR calculation
  • Total protein, albumin, lipid profile
  • Kidney biopsy with Congo red staining
  • Amyloid typing
  • Search for underlying disease: tests for inflammation, blood tests

Treatment and observation

The main principle is to target the source of amyloid. In the AA form, stable remission of the inflammatory disease is achieved; for periodic illness, the basic treatment is colchicine, prescribed for life. In the AL form, treatment is carried out by a hematologist, using regimens that suppress the plasma cell clone. At the same time, they protect the kidneys: control blood pressure, reduce protein loss, correct swelling with diuretics, and select a diet. All medications are prescribed by a doctor; Self-medication and dietary supplements for amyloidosis are not only useless, but also a dangerous waste of time.

  • Active treatment of the underlying disease
  • Colchicine for periodic illness as prescribed by a doctor
  • Hematological therapy for AL amyloidosis
  • Control of blood pressure and proteinuria
  • Diuretics and salt restriction for edema
  • Regular monitoring of kidney function
  • In the terminal stage - renal replacement therapy

Services and prices for this diagnosis

Based on official price lists of Tashkent clinics. The exact cost is determined after examination.

Frequently asked questions: Kidney amyloidosis

Is it possible to make a diagnosis without a biopsy?+
Amyloidosis can be suspected based on the clinical picture, but confirmed only morphologically. A biopsy is taken from the kidney or other available tissue, and the sample is stained with Congo red and typed. Without this, serious therapy cannot be prescribed.
Is renal amyloidosis hereditary?+
Some forms are hereditary, for example amyloidosis in periodic illness. But the most common variant in practice is secondary AA amyloidosis against the background of chronic inflammation; it is not inherited. An examination helps determine the type.
Are amyloid deposits reversible?+
They cannot be completely removed, however, with effective treatment of the underlying disease, new deposits stop forming, and the condition of the kidneys stabilizes. In some patients, proteinuria decreases.
Why is there such severe swelling with amyloidosis?+
The kidney loses a large amount of protein in the urine, which causes the level of albumin in the blood to drop and fluid to leak from the vessels into the tissues. This is called nephrotic syndrome. Treatment includes diuretics, salt restriction, and addressing the cause.
Which doctor should I go to first?+
To a nephrologist. He will evaluate the tests, arrange a biopsy and decide whether a rheumatologist or hematologist is needed. If the underlying disease is already known, treatment is carried out together with a specialized specialist.

The information on this page is for reference only and does not replace a doctor consultation. Only a qualified specialist can make a diagnosis and prescribe treatment after an in-person examination.

Where it is treated renal amyloidosis в Ташкенте

При стойкой протеинурии и отёках нужен нефролог, а для уточнения формы амилоидоза часто подключают ревматолога и гематолога. Clinics Ташкента, где ведут таких пациентов:

Tashkent, Mirabad district, st. Oybek, 34d
M Toshkent 🚶 550 m
M Oybek 🚶 850 m
M Kosmonavtlar 🚶 1.3 km
🚌 Nearest bus stop 🚶 150 m · buses: 22
Tashkent, Uchtepa district, st. Lutfiy 56d
M Olmazor 🚶 1.8 km
M Chilonzor 🚶 2.0 km
M Mirzo Ulug'bek 🚶 2.7 km
🚌 Nearest bus stop 🚶 20 m · buses: 2, 9Т, 13, 17T, 41, 56
Пн–Sat:08:00–17:00
Closed now
Tashkent, Almazar district, st. Usta Olim, 159d
M G'afur G'ulom 🚶 2.2 km
M Minor 🚶 2.3 km
M Abdulla Qodiriy 🚶 2.3 km
🚌 Nearest bus stop 🚶 190 m · buses: 5
Mon–Fri:09:00–17:00
Closed now
st. Magtymguly 105 (formerly Tarakkiyot), Yashnabad district, Tashkent Landmark: old TashM...
M Hamid Olimjon 🚶 1.5 km
M Mashinasozlar 🚶 1.5 km
M Ming O'rik 🚶 1.7 km
🚌 Nearest bus stop 🚶 240 m · buses: 14
Mon–Fri:08:00–18:00
Closed now
Tashkent, Yakkasaray district, st. Sh.Rustaveli, 109 A
M Oybek 🚶 2.4 km
M Kosmonavtlar 🚶 2.6 km
M Novza 🚶 2.9 km
🚌 Nearest bus stop 🚶 70 m · buses: 2, 11, 12, 40, 45, 47
Пн–Sat:08:00–17:30
Closed now
Tashkent, Olmazar Tumani, Kichik Khalka Yuli 5 "A" y
M Beruniy 🚶 500 m
M Tinchlik 🚶 1.1 km
M Chorsu 🚶 2.9 km
🚌 Nearest bus stop 🚶 190 m · buses: 31, 34
Mon–Fri:08:30–17:00
Closed now

ICD-10 code

Official international classification codes — these are used in medical records and statistics.

Other diseases: Urology

Book